Semax
10mg – Lyophilized *MUST BE RECONSTITUTED* Semax is a synthetic derivative of adrenocorticotrophic hormone, extensively studied for it
10mg – Lyophilized *MUST BE RECONSTITUTED* Semax is a synthetic derivative of adrenocorticotrophic hormone, extensively studied for its potential therapeutic benefits. Research suggests that Semax may improve immune function, protect neurons, and enhance cardiovascular health. Additionally, it has shown promise in pain control and promoting healthy blood clotting. Ideal for research applications, Semax offers a valuable tool for investigating advanced therapeutic strategies in neuroprotection, cardiovascular health, and immune system support.

Semax - 10mg
10mg – Lyophilized *MUST BE RECONSTITUTED* Semax is a synthetic derivative of adrenocorticotrophic hormone, extensively studied for it
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Semax / Selank - 10mg / 10mg
Semax – 10mg / Selank – 10mg Semax / Selank (20 mg) is a research-grade peptide formulation combining Semax (10 mg) and Selank (10 mg), deve

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References
- Semax - Peptide Center guide
- Functional Connectomic Approach to Studying Selank and Semax EffectsDoklady biological sciences : proceedings of the Academy of Sciences of the USSR, Biological sciences sections, 2020
- Semax peptide targets the μ opioid receptor gene Oprm1 to promote deubiquitination and functional recovery after spinal cord injury in female miceBritish journal of pharmacology, 2025
- Influence of the N-terminus acetylation of Semax, a synthetic analog of ACTH(4-10), on copper(II) and zinc(II) coordination and biological propertiesJournal of inorganic biochemistry, 2016
- The Potential of the Peptide Drug Semax and Its Derivative for Correcting Pathological Impairments in the Animal Model of Alzheimer's DiseaseActa naturae, 2025
- Semax and Pro-Gly-Pro activate the transcription of neurotrophins and their receptor genes after cerebral ischemiaCellular and molecular neurobiology, 2010
- The peptide semax affects the expression of genes related to the immune and vascular systems in rat brain focal ischemia: genome-wide transcriptional analysisBMC genomics, 2014
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